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Schizophrenia Bulletin Advance Access published online on September 11, 2008
Schizophrenia Bulletin, doi:10.1093/schbul/sbn116
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© The Author 2008. Published by Oxford University Press on behalf of the Maryland Psychiatric Research Center. All rights reserved. For permissions, please email: journals.permissions@oxfordjournals.org.

Introduction

Jim van Os1,2 and Robin Murray3
2 Division of Psychological Medicine, Institute of Psychiatry, London, UK, Maastricht University
3 Institute of Psychiatry

Keywords: schizophrenia / genetics / gene-environment interaction / risk factors / epidemiology / psychosis

The first 10% of the full text of this article appears below.

The search for the molecular genetic basis of schizophrenia has proven much more difficult than was initially thought. Indeed, the field is currently in a state of anxious self-examination with a range of explanations proposed for the failure to consistently replicate the putative susceptibility genes proposed in the early 2000s.1–4 One explanation that has received scant attention is gene-environment . . . [Full Text of this Article]

1 To whom correspondence should be addressed; Department of Psychiatry and Neuropsychology, Maastricht University Medical Centre, Maastricht, The Netherlands; tel: 31-43-3875443, fax: 31-43-3875444, e-mail: j.vanos@sp.unimaas.nl.


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